Search results for "PERIPHERAL NEUROPATHY"

showing 10 items of 55 documents

Effect of acetyl-l-carnitine in the treatment of diabetic peripheral neuropathy : A systematic review and meta-analysis

2017

Background/aim: Deficiency of acetyl-L-carnitine (ALC) and L-carnitine (LC) appears to play a role in peripheral diabetic neuropathy, although the evidence in humans is still limited. We conducted a systematic review and meta-analysis investigating the effect of ALC on pain and electromyographic parameters in people with diabetic neuropathy. Methods: A literature search in major databases, without language restriction, was undertaken. Eligible studies were randomized controlled trials (RCTs) or pre-and post-test studies. The effect of ALC supplementation on pain perception and electromyographic parameters in patients with diabetic neuropathy was compared vs. a control group (RCTs). The effe…

medicine.medical_specialtyDiabetic neuropathyDiabetePlaceboNerve conduction velocitylaw.inventionRATS03 medical and health sciencesGLUTAMATE0302 clinical medicineRandomized controlled triallawDiabetes mellitusMedicineQUALITYMeta-analysi030212 general & internal medicineUlnar nerveAdverse effectbusiness.industryDiabetesNERVE GROWTH-FACTORPAINmedicine.diseaseAcetyl-L-carnitine3. Good healthSurgeryNeuropathyAcetyl-L-carnitine; Diabetes; Meta-analysis; Neuropathy; Gerontology; Geriatrics and GerontologyMeta-analysisPeripheral neuropathyAnesthesia3121 General medicine internal medicine and other clinical medicineGeriatrics and GerontologybusinessGerontology030217 neurology & neurosurgeryCLINICAL-TRIALS
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Glucose regulation and pain in older people-The Helsinki Birth Cohort Study

2021

Aims: To assess if individuals with diabetes or prediabetes report more pain or have increased use of pain medication compared to normoglycaemic individuals. Methods: Using cross-sectional data, we studied 928 men and 1075 women from the Helsinki Birth Cohort Study in 2001-2004 at a mean age of 61.5 years. Glucose regulation was assessed with a 2-h 75 g oral glucose tolerance test, and applying World Health Organization criteria, participants were defined as having normoglycaemia, prediabetes (impaired fasting glucose or impaired glucose tolerance), newly diagnosed diabetes or previously diagnosed diabetes. Self-reported pain intensity and interference during the previous 4 weeks was estima…

Blood GlucoseMalePREDICTOREpidemiologyIMPACTEndocrinology Diabetes and MetabolismglukoosiaineenvaihduntaprediabetesDISEASECohort StudiesImpaired glucose tolerance0302 clinical medicinepain030212 general & internal medicinePrediabetesepidemiologiaPOPULATION2. Zero hungereducation.field_of_studyNutrition and DieteticsdiabetesDiabetesMiddle Aged3. Good healthPREVALENCEFemaleepidemiologyFamily Practiceikääntyneetmedicine.medical_specialtyPopulationPain030209 endocrinology & metabolismPrediabetic State03 medical and health sciencesInternal medicineDiabetes mellitusGlucose IntoleranceInternal MedicinemedicineHumansTOLERANCEeducationAgedbusiness.industryBeck Depression Inventorykipumedicine.diseaseImpaired fasting glucoseLIFEHIGH-RISKCross-Sectional StudiesGlucose3121 General medicine internal medicine and other clinical medicinekrooninen kipuBlood sugar regulationbusinessBody mass indexPrediabetesPERIPHERAL NEUROPATHY
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Peripheral Neuropathy in the Hypereosinophilic Syndrome: A Case Report

1989

We observed a patient with the hypereosinophilic syndrome that showed as a prominent clinical feature peripheral nerve dysfunction. The neuropathy evolved over 4 months and affected sensory and motor functions. Nerve conduction studies and EMG were compatible with axonal neuropathy. Nerve and muscle biopsies revealed severe axonal degeneration with neurogenic atrophy of muscle. Morphometry of peroneal nerve showed marked axonal loss, more prominent in large myelinated fibers. There was no evidence of vasculitis process. Neuropathy is produced by eosinophil-released substances exerting a neurotoxic effect through direct altered vascular endothelial permeability and local mast cell histamine …

Pathologymedicine.medical_specialtyBiopsyAxonal losschemistry.chemical_compoundEosinophiliaBiopsymedicineHumansAxonmedicine.diagnostic_testHypereosinophilic syndromebusiness.industryPeripheral Nervous System DiseasesMiddle Agedmedicine.diseaseMast cellPeripheral neuropathymedicine.anatomical_structureNeurologychemistryImmunologyFemaleNeurology (clinical)businessVasculitisHistamineEuropean Neurology
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The influence of local capsaicin treatment on small nerve fibre function and neurovascular control in symptomatic diabetic neuropathy.

2002

Topical treatment with capsaicin cream has been shown to be successful in the treatment of different symptomatic nerve disorders like diabetic neuropathy. Conflicting data exist on the effect of capsaicin on nerve function and neurovascular control especially in patients with diabetic neuropathy. The aim of this pilot study was to investigate the impact of topical capsaicin application on small nerve fibre function and neurovascular control. Capsaicin cream was applied to the feet of 13 patients with symptomatic diabetic neuropathy over a period of 8 weeks. Before and during the treatment period, we investigated the total symptoms score, the vibration, thermal (heat and cold) and pain perce…

MaleDiabetic neuropathyEndocrinology Diabetes and MetabolismAdministration TopicalSubstance PPilot Projectschemistry.chemical_compoundRoute of administrationEndocrinologyNerve FibersDiabetic NeuropathiesInternal MedicineMedicineHumansbusiness.industryGeneral MedicineMiddle Agedmedicine.diseaseNeurovascular bundlePeripheral neuropathymedicine.anatomical_structureDiabetes Mellitus Type 1chemistryDiabetes Mellitus Type 2CapsaicinAnesthesiaSensory ThresholdsNociceptorFemaleCapsaicinbusinessSensory nervePhytotherapyActa diabetologica
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A novel compound heterozygous mutation in GALC associated with adult-onset Krabbe disease: case report and literature review

2021

Krabbe disease (KD) is a rare autosomal recessive lipid storage leukodystrophy. It is caused by deficient enzyme activity resulting from mutations of the β-galactocerebrosidase (GALC) gene. KD is distinguished into subtypes based on the age of onset; these are early infantile, late infantile, juvenile, and adult-onset. We report a case of a 47-year-old Caucasian man with a 2-year history of muscle atrophy and weakness in both hands associated with pyramidal signs and mild spasticity in the lower limbs. An extensive work-up led this motor neuron disease-like disorder to be diagnosed as adult-onset KD. The patient was found to be compound heterozygous for two GALC mutations (p.G286D and p.Y49…

MaleCellular and Molecular NeuroscienceHeterozygoteMutationGeneticsHumansSettore MED/26 - NeurologiaMiddle AgedGenetics (clinical)Compound heterozygous mutation GALC Adult-onset Krabbe disease Peripheral neuropathyGalactosylceramidaseLeukodystrophy Globoid Cell
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Myo-, neuro-, gastrointestinal encephalopathy (MNGIE syndrome) due to partial deficiency of cytochrome-c-oxidase

1987

A 42-year-old woman had a 10-year history of external ophthalmoplegia, malabsorption resulting in chronic malnutrition, muscle atrophy and polyneuropathy. Computer tomography revealed hypodensity of her cerebral white matter. A metabolic disturbance consisted of lactic acidosis after moderate glucose loads with increased excretion of hydroxybutyric and fumaric acids. Post-mortem studies revealed gastrointestinal scleroderma as the morphological manifestation of her malabsorption syndrome, ocular and skeletal myopathy with ragged red fibers, peripheral neuropathy, vascular abnormalities of meningeal and peripheral nerve vessels. Biochemical examination of the liver and muscle tissues reveale…

AdultPathologymedicine.medical_specialtyMalabsorptionGastrointestinal DiseasesEncephalopathyRespiratory chainCytochrome-c Oxidase DeficiencyEyePathology and Forensic Medicine03 medical and health sciencesCellular and Molecular Neuroscience0302 clinical medicineMuscular DiseasesMitochondrial myopathymedicineHumansMuscular dystrophy030304 developmental biology2. Zero hungerBrain Diseases0303 health sciencesbusiness.industryPeripheral Nervous System DiseasesSyndromemedicine.diseaseMitochondria MusclePeripheral neuropathyLactic acidosisFemaleNeurology (clinical)businessPolyneuropathy030217 neurology & neurosurgeryActa Neuropathologica
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Peripheral blood levels of CXCL10 are a useful marker for diabetic polyneuropathy in subjects with type 2 diabetes.

2020

BACKGROUND Diabetic peripheral neuropathy (DPN) is a chronic complication of diabetes mellitus associated with high morbidity and mortality. Major risk factors for DPN include metabolic changes, duration of diabetes, nerve ischaemia and derangements in regeneration and nerve repair programmes. Chemokines have been previously implicated in the pathogenesis of various neuropathies and neuropathic pain processes. The aim of this pilot study was to evaluate the association between the plasma levels of chemokines (CXCL9, CXCL10 and CXCL11) in the presence of DPN in a cohort of type 2 diabetes (T2D) patients. MATERIALS AND METHODS We studied 73 patients with T2D: 36 with DPN and 37 without DPN. D…

medicine.medical_specialtybusiness.industryPilot ProjectsGeneral MedicineType 2 diabetesmedicine.diseaseGastroenterologyChemokine CXCL10Peripheral neuropathyDiabetes Mellitus Type 2Diabetic NeuropathiesRisk FactorsInternal medicineDiabetes mellitusNeuropathic painmedicineCXCL10CXCL9HumansCXCL11ComplicationbusinessBiomarkersInternational journal of clinical practiceREFERENCES
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Cisplatin-induced peripheral neuropathy: neuroprotection by erythropoietin without affecting tumour growth

2007

This study examined the dose-dependent efficacy of erythropoietin (EPO) for preventing and/or treating cisplatin (CDDP) induced peripheral neurotoxicity (CINP), and its influence on tumour treatment and growth. Rats received eight intraperitoneal (ip) injections of 2 mg/kg CDDP twice weekly. EPO co-administered (50 or 10 microg/kg ip, three times/week) had a dose-dependent effect, partially preventing CINP, but 0.5 microg/kg ip was not effective. The neuroprotective effect lasted at least 5 weeks after the last dose of EPO and CDDP. In addition, EPO (50 microg/kg ip three times/week) after the last injection of CDDP still induced a significant recovery of CINP. In a separate experiment in r…

medicine.medical_specialtyCancer ResearchPeripheral neuropathyNeural ConductionNeurophysiologyAntineoplastic AgentsHindlimbHematocritNeuroprotectionAntineoplastic AgentInternal medicinemedicinePathologyAnimalsRats WistarErythropoietinCisplatincisplatin; Erythropoietin; peripheral neuropathy; tumor growthmedicine.diagnostic_testDose-Response Relationship Drugbusiness.industryAnimalNeurotoxicityPeripheral Nervous System DiseasesMammary Neoplasms ExperimentalTumour growthHematologymedicine.diseaseRatsHindlimbDose–response relationshipPeripheral neuropathyEndocrinologyOncologyHematocritErythropoietinRatFemalePeripheral Nervous System DiseaseCisplatinbusinessCell Divisionmedicine.drug
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Complexity of the Hereditary Motor and Sensory Neuropathies

2015

Early-onset hereditary motor and sensory neuropathies are rare diseases representing a broad clinical and genetic spectrum. Without a notable familial history, the clinical diagnosis is complicated because acquired causes of peripheral neuropathy, such as inflammatory neuropathies, neuropathies with toxic causes, and nutritional deficiencies, must be considered. We examined the clinical, electrophysiological, and pathologic manifestations of a boy with an initial diagnosis of chronic inflammatory demyelinating polyneuropathy. The progression of the disease despite treatment led to a suspicion of hereditary motor and sensory neuropathy. Genetic testing revealed the presence of the MPZ p.D90…

MalePathologymedicine.medical_specialtyChronic inflammatory demyelinating polyneuropathySensory systemDiseaseBioinformaticsSural NervemedicineHumansGenetic testingmedicine.diagnostic_testbusiness.industrymedicine.diseasePhenotypePeripheral neuropathyChild PreschoolClinical diagnosisMutationPediatrics Perinatology and Child HealthMutation (genetic algorithm)Disease ProgressionNeurology (clinical)Hereditary Sensory and Motor NeuropathybusinessHereditary motor and sensory neuropathyMyelin P0 ProteinHeLa CellsJournal of Child Neurology
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Diabetic Peripheral Neuropathy is Associated With Diabetic Kidney Disease and Cardiovascular Disease:The Silesia Diabetes-Heart Project

2023

Microvascular complications of diabetes seem to be clustered and put patients at higher risk of developing cardiovascular disease (CVD). This was a questionnaire-based study designed to screen for the presence of diabetic peripheral neuropathy (DPN), defined as the score in the Michigan Neuropathy Screening Instrument (MNSI) above 2, and to evaluate its association with other complication of diabetes, including CVD. There were 184 patients included into the study. The prevalence of DPN in the study group was 37.5%. The regression model analysis revealed that the presence of DPN was significantly associated with the presence of diabetic kidney disease (DKD) (P = 0.0034;) and patient's age (P…

Michigan Neuropathy Screening Instrument; cardiovascular disease; diabetes mellitus; diabetic kidney disease; diabetic peripheral neuropathyGeneral MedicineCardiology and Cardiovascular Medicine
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